Main Content
Publications
The Conserved Chromatin Remodeler SMARCAD1 Interacts with TFIIIC and Architectural Proteins in Human and Mouse. Parysatis Sachs, Philipp Bergmaier, Katrin Treutwein, and Jacqueline E. Mermoud Genes 2023; https://doi.org/10.3390/genes14091793 |
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The Role of the m6A RNA Methyltransferase METTL16 in Gene Expression and SAM Homeostasis. Jacqueline E. Mermoud Genes 2022, https://doi.org/10.3390/genes13122312 |
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Roles and regulation of endogenous retroviruses in pluripotency and early development. Sharon Schlesinger, Miguel R. Branco and Mermoud J.E. Stem Cell Epigenetics, Volume 17 in Translational Epigenetics p 155-186 (2020) |
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SMARCAD1 ATPase activity is required to silence endogenous retroviruses in embryonic stem cells. |
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The CUE1 domain of the SNF2-like chromatin remodeler SMARCAD1 mediates its association with KRAB-associated protein 1 (KAP1) and KAP1 target genes. |
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Keeping chromatin quiet: how nucleosome remodeling restores heterochromatin after replication. |
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Maintenance of silent chromatin through replication requires SWI/SNF-like chromatin remodeler SMARCAD1. · Previewed in Mol Cell. 42, 267-269, by Jasencakova and Groth |
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Neurite-like structures induced by mevalonate pathway blockade are due to the stability of cell adhesion foci and are enhanced by the presence of APP. |
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Epigenomes under scrutiny. Report on the 3rd UK Stem Cell Meeting. |
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Global hypomethylation of the genome in XX ES cells. |
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Polycomb group proteins Ring1A/B link ubiquitylation of histone H2A to heritable gene silencing and X inactivation. |
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Histone H3 lysine 9 methylation occurs rapidly at the onset of random X chromosome inactivation. |
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Xist expression and macroH2A1.2 localisation in mouse primordial and pluripotent embryonic germ cells. |
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Histone H3 lysine 9 methylation is an epigenetic imprint of facultative heterochromatin. |
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Centrosomal association of histone macroH2A1.2 in embryonic stem cells and somatic cells. |
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Histone macroH2A1.2 relocates to the inactive X chromosome after initiation and propagation of X inactivation. |
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Cis acting elements distinct from the 5´ splice site promote U1-independent pre-mRNA splicing. |
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Uncovering the role of Ser/Thr protein phosphorylation in nuclear pre-mRNA splicing. |
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Regulation of mammalian spliceosome assembly by a protein phosphorylation mechanism. |
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Ser/Thr-specific protein phosphatases are required for both catalytic steps of pre-mRNA splicing. |
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